Question

Choose the correct answer from the four options given. During blood coagulation, thromboplastin is released by?
A.RBC
B.Clumped platelets and and damaged tissues
C.Blood plasma
D.Leucocyte

Answer 1

The extrinsic pathway begins with a substance called tissue factor (tissue thromboplastin) released by damaged blood vessels and surrounding tissues. In the presence of other plasma proteins (clotting factors) and calcium ions , this leads to the activation of a protein called factor X.

Complete answer:
- RBCs express phosphatidylserine and microparticles, supporting thrombin age.They interface with platelets, endothelial cells, and fibrinogen, and these coordinated efforts lead their breaker into the thrombi. The presence of RBCs in bunches covers plasmin age and reduces cluster breaking down. Lessening blood cluster RBC substance would enliven clump's objective. Taking everything into account, RBCs are significant supplements of the mind boggling responses of cluster arrangement.

- Injured tissue (vessel)discharges thromboplastin and gathered platelets discharge platelet factors.Both thromboplastin and platelet factors respond with coagulation factors in the plasma to make a prothrombin activator.

- Thromboplastin is a plasma protein helping blood coagulation through catalyzing the change of prothrombin to thrombin. It is a perplexing compound that is found in cerebrum, lung, and different tissues and particularly in the blood platelets. Heparin is delivered by the pole cells and blood basophils. Fibrinogen is delivered by the hepatic parenchymal cell.

-Leukocytes are a dynamic and vagrant segment of the intrinsic insusceptible framework that intercede a quick reaction to procoagulant improvements. Restricted intravascular coagulation including initiated leukocytes probably developed as a versatile component to advance the goal of disease when injury and pestilences represented most of human passions.

So the Correct option is B

Note: RBCs may contribute in a few different ways. In polycythemia, RBCs increment blood thickness and marginate platelets toward the endothelium. The expanded incidence of thrombosis is also connected with hemolytic anemia, especially with sickle cell disease and paroxysmal nocturnal hemoglobinuria.